Primary Hyperaldosteronism Revealed by Torsade de Pointes: Case Report

Boucetta Abdellah *

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

O. Saleh

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

Abdelani Badr

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

Abdenasser Drighil

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

Ghali Benoun

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

Rachida Habbal

Service of Cardiology, CHU Ibn Rochd of Casablanca, Morocco.

*Author to whom correspondence should be addressed.


Abstract

Introduction: Primary hyperaldosteronism is a disorder characterised by the excessive production of aldosterone by the adrenal glands. It typically presents as elevated blood pressure and low blood potassium levels. However, the clinical manifestation can be significantly detrimental when metabolic disorders and hypertension result in severe arrhythmias.

Case Report: This is a report of a case of primary hyperaldosteronism in a 37-year-old male who was admitted to hospital with syncope, palpitations and dyspnoea. A clinical examination of the patient revealed an unstable condition with tachycardia (180 beats per minute). An electrocardiogram showed evidence of torsade de pointes, which necessitated defibrillation and a return to sinus rhythm. Transthoracic echocardiography demonstrated evidence of ventriculo atrial remodelling and diastolic dysfunction.

Conclusion: Therefore, when faced with life-threatening arrhythmias, it is imperative to induce primary hyperaldosteronism in the context of etiological research, especially when hypokalemia and hypertension exist.

Keywords: Primary hyperaldosteronism, torsade de pointes, case report


How to Cite

Abdellah, Boucetta, O. Saleh, Abdelani Badr, Abdenasser Drighil, Ghali Benoun, and Rachida Habbal. 2024. “Primary Hyperaldosteronism Revealed by Torsade De Pointes: Case Report”. Asian Journal of Cardiology Research 7 (1):189-94. https://journalajcr.com/index.php/AJCR/article/view/218.

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